PharmaFreak Anabolic Freak 2.0 180 Caps
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- The vitamin combination of B6, folate and B12 is shown to significantly increase nitric oxide (NO) and blood flow to working muscles.
- Rhodiola rosea is shown to reduce stress and block cortisol production.
- A clinically backed dosage of 3,120 mg of d-aspartic acid calcium chelate supports natural testosterone production.
D-Aspartic Acid Calcium Chelate
To get the most out of d-aspartic acid, you must use a form that’s highly soluble and bioavailable so it can get to the testes and pituitary gland to do its job and stimulate more testosterone release. Regular sodium d-aspartic acid has very bad solubility, which means poor bioavailability. By creating a calcium chelate, the solubility of DAA is increased by over 200%, which means significantly greater bioavailability and uptake into the testes and pituitary gland. The end result is greater total testosterone production.
A ground-breaking study published in the journal Reproductive Biology and Endocrinology examined the effects of DAA supplementation on testosterone secretion in healthy young men. In the study, subjects were given a daily dose of 3,120 mg of DAA for 12 days whereas the control group was given a placebo. After 12 days, the subjects supplementing with DAA experienced, on average, a 42% increase in testosterone release and a 33% increase in luteinizing hormone. The thing that makes these results even more impressive is the study design was gold-standard, double-blind and placebo-controlled.
Vitamin B6, Folic Acid & Vitamin B12
Vitamins B6, folic acid and B12 can support healthy nitric oxide (NO) production and blood flow. Some research suggests that supplementation with vitamins B6, folic acid and B12 can help reduce the concentration of homocysteine in the bloodstream. Homocysteine is a nonprotein amino acid, and excessive levels can impede healthy NO production and vascular blood flow.
Resveratrol can block the aromatase enzyme and help prevent the conversion of testosterone to estrogen. Resveratrol also inhibits gene transcription of the aromatase enzyme, blocking estrogen production at the gene level.